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1.

001-es BibID:BIBFORM039245
Első szerző:Ács Géza (traumatológus)
Cím:Differential activation and desensitization of sensory neurons by resiniferatoxin / Acs, G., Biro, T., Acs, P., Modarres, S., Blumberg, P. M.
Dátum:1997
ISSN:0270-6474
Megjegyzések:Recently, with use of rat dorsal root ganglion (DRG) neurons we have been able to dissociate the binding affinities of vanilloids from their potencies to induce Ca-45 uptake, which suggests the existence of distinct classes of the vanilloid receptor (Acs et al., 1996). In the present study, we have demonstrated that the ultrapotent capsaicin analog resiniferatoxin (RTX) desensitized rat DRG neurons to the subsequent induction of Ca-45 uptake by capsaicin and RTX with affinity and cooperativity similar to that found for [H-3]RTX binding, contrasting with a similar to 10-fold weaker potency and lack of cooperativity to induce Ca-45 uptake. Likewise, the competitive antagonist capsazepine inhibited RTX-induced desensitization with potency similar to that for inhibition of specific [H-3]RTX binding, whereas the potency of capsazepine was similar to 10-fold higher for inhibiting RTX-induced Ca-45 uptake. Finally, the noncompetitive antagonist ruthenium red inhibited both the RTX-induced desensitization and Ca-45 uptake but showed similar to 60-fold selectivity for inhibiting RTX-induced desensitization. The RTX-induced desensitization was not associated with loss of specific [H-3]RTX binding, suggesting lack of gross cell toxicity. In contrast to RTX, capsaicin caused desensitization with a potency corresponding to that for Ca-45 uptake and did so in a noncooperative manner. Unlike the RTX-induced desensitization, the desensitization by capsaicin was blocked by ruthenium red only at doses that blocked Ca-45 uptake and depended on external calcium, Our findings provide further support for the existence of vanilloid receptor subtypes on DRG neurons with distinct pharmacology and distinct patterns of desensitization.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal of Neuroscience. - 17 : 14 (1997), p. 5622-5628. -
További szerzők:Bíró Tamás (1968-) (élettanász) Ács Péter Modarres, Shayan Blumberg, Peter M.
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2.

001-es BibID:BIBFORM029094
Első szerző:Antal Miklós (orvos, anatómus)
Cím:Expression of hyperpolarization-activated and cyclic nucleotide-gated cation channel subunit 2 in axon terminals of peptidergic nociceptive primary sensory neurons in the superficial spinal dorsal horn of rats / Antal, M., Papp, I., Bahaerguli, N., Veress, G., Vereb, G.
Dátum:2004
Megjegyzések:Hyperpolarization-activated cyclic nucleotide-gated cation channel proteins (HCN1-4), which are potentially able to modulate membrane excitability, are abundantly expressed by neurons in spinal dorsal root ganglia (DRG). In the present experiment, we investigated whether HCN2 protein is confined exclusively to the perikarya of DRG neurons or is transported from the somata to the central axons of DRG neurons that terminate in the spinal dorsal horn. Using immunohistochemical methods, we have demonstrated that laminae I-IIo of the superficial spinal dorsal horn of the adult rat spinal cord show a strong punctate immunoreactivity for HCN2. Dorsal rhizotomy resulted in a complete loss of immunostaining in the dorsal horn, suggesting that HCN2 is confined to axon terminals of primary afferents. In double labelling immunohistochemical studies, we have also shown that HCN2 widely co-localizes with calcitonin gene-related peptide, but is almost completely segregated from isolectin-B4 binding, indicating that HCN2 is primarily expressed in peptidergic nociceptive primary afferents. The expression of HCN2 in central terminals of peptidergic primary afferents was also verified with electron microscopy. Utilizing the pre-embedding nanogold method, we found that HCN2 is largely confined to axon terminals with dense-core vesicles. Within these terminals, some of the silver grains marking the accurate location of HCN2 molecules were associated with the cell membrane, and others were scattered in the axoplasm. Within the cell membrane, HCN2 was found almost exclusively in extrasynaptic locations. The results suggest that HCN2 may contribute to the modulation of membrane excitability of nociceptive primary afferent terminals in the spinal dorsal horn.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:The European Journal of Neuroscience 19 : 5 (2004), p. 1336-1342. -
További szerzők:Papp Ildikó (1976-) (biológus) Bahaerguli, Niyazi Veress Gábor (1971-) (neurobiológus) Vereb György (1965-) (biofizikus, orvos)
Internet cím:Intézményi repozitóriumban (DEA) tárolt változat
elektronikus változat
DOI
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3.

001-es BibID:BIBFORM004385
Első szerző:Antal Miklós (orvos, anatómus)
Cím:Numbers, densities, and colocalization of AMPA- and NMDA-type glutamate receptors at individual synapses in the superficial spinal dorsal horn of rats / Miklós Antal, Yugo Fukazawa, Mária Eördögh, Dóra Muszil, Elek Molnár, Makoto Itakura, Masami Takahashi, Ryuichi Shigemoto
Dátum:2008
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:The Journal of Neuroscience. - 28 : 39 (2008), p. 9692-9701. -
További szerzők:Fukazawa, Yugo Eördögh Mária Muszil Dóra Molnár Elek Itakura, Makoto Takahashi, Masami Shigemoto, Ryuichi
Internet cím:elektronikus változat
DOI
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4.

001-es BibID:BIBFORM103565
035-os BibID:(Scopus)85137531133 (WoS)000849139400001
Első szerző:Aranyi Sándor Csaba (programtervező informatikus)
Cím:Topological dissimilarities of hierarchical resting networks in type 2 diabetes mellitus and obesity / Aranyi Sándor Csaba, Képes Zita, Nagy Marianna, Opposits Gábor, Garai Ildikó, Káplár Miklós, Emri Miklós
Dátum:2023
ISSN:0929-5313
Megjegyzések:Type 2 diabetes mellitus (T2DM) is reported to cause widespread changes in brain function, leading to cognitive impairments. Research using resting-state functional magnetic resonance imaging data already aims to understand functional changes in complex brain connectivity systems. However, no previous studies with dynamic causal modelling (DCM) tried to investigate large-scale effective connectivity in diabetes. We aimed to examine the differences in large-scale resting state networks in diabetic and obese patients using combined DCM and graph theory methodologies. With the participation of 70 subjects (43 diabetics, 27 obese), we used cross-spectra DCM to estimate connectivity between 36 regions, subdivided into seven resting networks (RSN) commonly recognized in the literature. We assessed group-wise connectivity of T2DM and obesity, as well as group differences, with parametric empirical Bayes and Bayesian model reduction techniques. We analyzed network connectivity globally, between RSNs, and regionally. We found that average connection strength was higher in T2DM globally and between RSNs, as well. On the network level, the salience network shows stronger total within-network connectivity in diabetes (8.07) than in the obese group (4.02). Regionally, we measured the most significant average decrease in the right middle temporal gyrus (-0.013 Hz) and the right inferior parietal lobule (-0.01 Hz) relative to the obese group. In comparison, connectivity increased most notably in the left anterior prefrontal cortex (0.01 Hz) and the medial dorsal thalamus (0.009 Hz). In conclusion, we find the usage of complex analysis of large-scale networks suitable for diabetes instead of focusing on specific changes in brain function.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
folyóiratcikk
Type 2 diabetes mellitus
Obesity
Effective connectivity
Dynamic causal modelling
Graph theory
Megjelenés:Journal Of Computational Neuroscience. - 51 : 1 (2023), p. 71-86. -
További szerzők:Képes Zita (1991-) (orvos) Nagy Marianna (1987-) (orvosdiagnosztikai képalkotó) Opposits Gábor (1974-) (fizikus, szoftver fejlesztő) Garai Ildikó (1966-) (radiológus) Káplár Miklós (1965-) (belgyógyász, diabetológus) Emri Miklós (1962-) (fizikus)
Pályázati támogatás:2017-1.2.1-NKP-2017-00002
Egyéb
TKP2021-NKTA-34
Egyéb
GINOP-2.1.1-15-2015-00609
GINOP
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5.

001-es BibID:BIBFORM028132
Első szerző:Asztalos Zoltán
Cím:Protein phosphatase 1-deficient mutant Drosophila is affected in habituation and associative learning / Zoltán Asztalos, Jörg von Wegerer, Gerald Wustmann, Viktor Dombrádi, János Gausz, Hanns-Christof Spatz, Peter Friedrich
Dátum:1993
ISSN:0270-6474
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
protein phosphatase 1
Drosophila
Megjelenés:Journal Of Neuroscience. - 13 : 3 (1993), p. 924-930. -
További szerzők:Wegerer, Jörg von Wustmann, Gerald Gausz János Spatz, Hanns-Christof Friedrich Péter Dombrádi Viktor (1953-) (biokémikus)
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6.

001-es BibID:BIBFORM054375
Első szerző:Bánki Eszter
Cím:Molecular Mechanisms Underlying the Nephroprotective Effects of PACAP in Diabetes / Eszter Banki, Krisztina Kovacs, Daniel Nagy, Tamas Juhasz, Peter Degrell, Katalin Csanaky, Peter Kiss, Gabor Jancso, Gabor Toth, Andrea Tamas, Dora Reglodi
Dátum:2014
ISSN:0895-8696
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal of Molecular Neuroscience 54 : 3 (2014), p. 300-309. -
További szerzők:Kovács Krisztina (Pécs) Nagy Dániel Juhász Tamás (1976-) (biológus, orvosbiológus) Degrell Péter Csanaky Katalin Kiss Péter (Pécs) Jancsó Gábor Tóth Gábor Tamás Andrea (Idegtudomány) (Pécs) Reglődi Dóra (Idegtudományok)
Pályázati támogatás:K104984
OTKA
108596
OTKA
TÁMOP-4.2.2.A-11/1/KONV-2012-0024
TÁMOP
Bolyai Ösztöndíj
Egyéb
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7.

001-es BibID:BIBFORM070542
Első szerző:Bardóczi Zsuzsanna
Cím:Glycinergic input to the mouse basal forebrain cholinergic neurons / Bardóczi Z., Pál B., Kőszeghy Á., Wilheim T., Watanabe M., Záborszky L., Liposits Z., Kalló I.
Dátum:2017
Megjegyzések:The basal forebrain (BF) receives afferents from brain stem ascending pathways, which has been implicated first by Moruzzi and Magoun (Moruzzi and Magoun, 1949) to induce forebrain activation and cortical arousal/waking behavior; however, it is very little known about how brain stem inhibitory inputs affect cholinergic functions. In the current study, glycine, a major inhibitory neurotransmitter of brain stem neurons, and gliotransmitter of local glial cells, was tested for potential interaction with basal forebrain cholinergic (BFC) neurons in male mice. In the BF, glycine receptor ? subunit-immunoreactive (GlyR?-IR) sites were localized in choline acetyltransferase (ChAT)-IR neurons. Glycine's effect on BFC neurons was demonstrated by bicuculline-resistant, strychnine-sensitive spontaneous inhibitory postsynaptic currents (IPSCs; 0.81 ? 0.25 *10-1 Hz) recorded in whole cell conditions. Potential neuronal, as well as glial sources of glycine were indicated in the extracellular space of cholinergic neurons by glycine transporter 1 and 2 (GLYT1 and 2)-IR processes found in apposition to ChAT-IR cells. Ultrastructural analyses identified synapses of GLYT2-positive axon terminals on ChAT-IR neurons, as well as GLYT1-positive astroglial processes, which were localized in the vicinity of synapses of ChAT-IR neurons. The brain stem raphe magnus was determined to be a major source of glycinergic axons traced retrogradely from the BF. Our results indicate a direct effect of glycine on BFC neurons. Furthermore, the presence of high levels of plasma membrane glycine transporters in the vicinity of cholinergic neurons suggests a tight control of extracellular glycine in the BF.SIGNIFICANCE STATEMENTBFC neurons receive various activating inputs from specific brain stem areas, and channel this information to the cortex via multiple projections. So far very little is known about inhibitory brain stem afferents to the BF. The current study established glycine as a major regulator of BFC neurons by (1) identifying glycinergic neurons in the brain stem projecting to the BF, (2) showing GlyR?-IR sites in ChAT-IR neurons, (3) demonstrating GLYT2-positive axon terminals synapsing on ChAT-IR neurons, and (4) localizing GLYT1-positive astroglial processes in the vicinity of synapses of ChAT-IR neurons. Glycine's effect on BFC neurons was demonstrated by bicuculline-resistant, strychnine-sensitive spontaneous IPSCs recorded in whole cell conditions.
Tárgyszavak:Természettudományok Biológiai tudományok idegen nyelvű folyóiratközlemény külföldi lapban
basal forebrain
glycinergic
Megjelenés:The Journal of Neuroscience 37 : 39 (2017), p. 9534-9549. -
További szerzők:Pál Balázs (1975-) (élettanász) Kőszeghy Áron (1983-) (Ph.D hallgató, élettanász) Wilheim Tamás Watanabe, Masahiko Záborszky László Liposits Zsolt Kalló Imre
Pályázati támogatás:KTIA_13_NAP-A-I/10
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8.

001-es BibID:BIBFORM040762
Első szerző:Batta József Tamás (fül-orr-gégész)
Cím:Regulation of the lateral wall stiffness by acetylcholine and GABA in the outer hair cells of the guinea pig / Batta T. J., Panyi Gy., Szucs A., Sziklai I.
Dátum:2004
ISSN:0953-816X
Megjegyzések:Acetylcholine (ACh) and GABA, the main neurotransmitters of the efferent innervation of the outer hair cells (OHCs), are assumed to regulate the efficacy of the cochlear amplifier through a variety of mechanisms. The recently described stretch-induced changes of the lateral wall stiffness (regulatory stiffness response) and the stretch-induced slow cell motility of OHCs may be important regulatory mechanisms in this process. We found that ACh in cochleobasal OHCs significantly reduces the stiffness of the lateral wall but increases the regulatory stiffness response and stretch-induced slow cell motility. Qualitatively similar cellular responses were evoked by GABA in cochleoapical OHCs. The effects of ACh could be inhibited by strychnine, the specific inhibitor of the alpha(9) ACh receptors expressed in OHCs, whereas the effects of GABA could be blocked by bicuculline, a specific GABA(A) receptor antagonist. In the absence of extracellular Ca(2+) the effects of ACh and GABA on the regulatory stiffness response were reduced, indicating the involvement of Ca(2+) in the control of this process. Based on our results we suggest that efferent innervation protects the organ of Corti against high sound intensities and supports adaptation by modification of the micromechanical properties of OHCs. This could be governed by ACh and GABA indirectly, via the potentiation of stretch-induced cell shortening in a Ca(2+)-dependent manner, rather than by a direct stiffness regulation-related mechanism.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:European Journal Of Neuroscience. - 20 : 12 (2004), p. 3364-3370. -
További szerzők:Panyi György (1966-) (biofizikus) Szűcs Attila (1970-) (fül-orr-gégész) Sziklai István (1954-) (fül-orr-gégész)
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9.

001-es BibID:BIBFORM050040
Első szerző:Bhattacharya, Anirban
Cím:Potentiation of inhibitory synaptic transmission by extracellular ATP in rat suprachiasmatic nuclei / Anirban Bhattacharya, Vojtech Vavra, Irena Svobodova, Zdena Bendova, Gyorgy Vereb, Hana Zemkova
Dátum:2013
Megjegyzések:The hypothalamic suprachiasmatic nuclei (SCN), the circadian master clock in mammals, releases ATP in a rhythm, but the role of extracellular ATP in the SCN is still unknown. In this study, we examined the expression and function of ATP-gated P2X receptors (P2XRs) in the SCN neurons of slices isolated from the brain of 16- to 20-day-old rats. Quantitative RT-PCR showed that the SCN contains mRNA for P2X 1-7 receptors and several G-protein-coupled P2Y receptors. Among the P2XR subunits, the P2X2 > P2X7 > P2X4 mRNAs were the most abundant. Whole-cell patch-clamp recordings from SCN neurons revealed that extracellular ATP application increased the frequency of spontaneous GABAergic IPSCs without changes in their amplitudes. The effect of ATP appears to be mediated by presynaptic P2X2Rs because ATPgammaS and 2MeS-ATP mimics, while the P2XR antagonist PPADS blocks, the observed enhancement of the frequency of GABA currents. There were significant differences between two SCN regions in that the effect of ATP was higher in the ventrolateral subdivision, which is densely innervated from outside the SCN. Little evidence was found for the presence of P2XR channels in somata of SCN neurons as P2X2R immunoreactivity colocalized with synapsin and ATP-induced current was observed in only 7% of cells. In fura-2 AM-loaded slices, BzATP as well as ADP stimulated intracellular Ca(2+) increase, indicating that the SCN cells express functional P2X7 and P2Y receptors. Our data suggest that ATP activates presynaptic P2X2Rs to regulate inhibitory synaptic transmission within the SCN and that this effect varies between regions.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Adenosine
Adenosine Triphosphate
Animals
Animals,Newborn
article
Biophysical Phenomena
Brain
Calcium
Cells
Cells,Cultured
cytology
Dose-Response Relationship,Drug
drug effects
Excitatory Amino Acid Antagonists
EXPRESSION
gamma-Aminobutyric Acid
Gene Expression Regulation
genetics
In Vitro
Male
metabolism
Neural Inhibition
Neurons
Patch-Clamp Techniques
pharmacology
physiology
Platelet Aggregation
Platelet Aggregation Inhibitors
Purinergic Agents
Rats
Rats,Wistar
Receptors,Purinergic P2X
Research
Research Support
rna
RNA,Messenger
Sodium
Sodium Channel Blockers
Support
Suprachiasmatic Nucleus
Synaptic Potentials
Synaptic Transmission
Tetrodotoxin
Megjelenés:The Journal of Neuroscience. - 33 : 18 (2013), p. 8035-8044. -
További szerzők:Vavra, Vojtech Svobodova, Irena Bendova, Zdena Vereb György (1965-) (biofizikus, orvos) Zemkova, Hana
Internet cím:DOI
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10.

001-es BibID:BIBFORM003873
Első szerző:Boros Ákos
Cím:Changes in the expression of PACAP-like compounds during the embryonic development of the earthworm eisenia fetida / Akos Boros, Dora Reglodi, Zsofia Herbert, Gabor Kiszler, Jozsef Nemeth, Andrea Lubics, Peter Kiss, Andrea Tamas, Seiji Shioda, Kouhei Matsuda, Edit Pollak, Laszló Molnar
Dátum:2008
Megjegyzések:Pituitary adenylate cyclase-activating polypeptide (PACAP) is expressed at very early stages in the vertebrate nervous system, and its functions in the embryonic development have been shown by various studies. PACAP is an extremely conserved molecule in phylogeny; however, little is known about its presence and functions in invertebrates. Our previous studies have shown the occurrence of PACAP-like immunoreactivity in the invertebrate nervous system. The aim of this study was to investigate the presence and localization of PACAP-like compounds during the embryonic development of earthworms from cocoon deposition to hatching using immunological methods (radioimmunoassay, dot blot, immunohistochemistry). PACAP-like immunoreactive compounds were detected at very early stages of the embryonic development of the earthworm Eisenia fetida. No significant changes were observed during the early stages in the developing embryo, but a marked increase occurred before hatching. In contrast, during the embryonic development, the level of PACAP-like compounds gradually decreased in cocoon fluids. Immunohistochemistry revealed the presence of PACAP-like immunoreactive cell bodies and processes in the developing body wall, prostomium, pharyngeal wall, and central nervous system. Cells located in the body wall correspond to putative progenitor cells of primary sensory cells. In the present study, we also showed that the clitellum (reproductive organ) of sexually mature worms contained significantly higher levels of PACAP-like immunoreactivity than other regions of the same animals or the clitellar region of a non-reproducing animal. In summary, these observations provide a morphological basis and suggest a role of PACAP(-like peptides) in the reproductive and developmental functions of invertebrates.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Dot blot
Clitellum
Cocoon
Earthworm
embryo
Primary sensory cells
Radioimmunoassay
Megjelenés:Journal of molecular neuroscience. - 36 : 1-3 (2008), p. 157-165. -
További szerzők:Reglődi Dóra (Idegtudományok) Herbert Zsófia Kiszler Gábor Németh József (1954-) (vegyész, analitikus) Lubics Andrea (Pécs) Kiss Péter Tamás Andrea (Idegtudomány) (Pécs) Shioda, Seiji Matsuda, Kouhei Pollák Edit Molnár László
Internet cím:elektronikus változat
DOI
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11.

001-es BibID:BIBFORM013274
Első szerző:Brubel Réka
Cím:Changes in the Expression of Pituitary Adenylate Cyclase-Activating Polypeptide in the Human Placenta during Pregnancy and Its Effects on the Survival of JAR Choriocarcinoma Cells/ R. Brubel, A. Boronkai, D. Reglodi, B. Racz, J. Nemeth, P. Kiss, A. Lubics, G. Toth, G. Horvath, T. Varga, D. Szogyi, E. Fonagy, J. Farkas, A. Barakonyi, Sz. Bellyei, L. Szereday, M. Koppan, A. Tamas
Dátum:2010
Megjegyzések:Pituitary adenylate cyclase-activating polypeptide(PACAP), a neuropeptide with survival-promotingactions, has been observed in endocrine organs and isthought to play a role in reproductive functions, includingpregnancy. PACAP occurs in two forms, 27 and 38 aminoacid residues, with PACAP38 being the predominant formin human tissues. In the present study, we determined theconcentrations of PACAP38 and PACAP27 in firsttrimesterand full-term human placentas using radioimmunoassay.We found high levels of PACAP38 and lowerlevels of PACAP27 in different parts of the full-term humanplacenta. PACAP38 content increased in the placentaduring pregnancy, both on the maternal side and on thefetal side. The effects of PACAP on the survival of JARhuman choriocarcinoma cells were investigated using flowcytometry and MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetrazolium bromide) cell viability assay in cellsexposed to the widely used chemotherapeutic agentmethotrexate (MTX). It was found that PACAP neitherinfluenced the survival of JAR cytotrophoblast cells noraffected cellular response to the death-inducing effect of thechemotherapeutic agent MTX. The present observationsfurther support the significance of PACAP in the humanplacenta. The observation that PACAP did not influence theeffects of MTX may have future clinical importance,showing that PACAP does not decrease the effects ofcertain chemotherapeutic agents.Keywords JAR . Choriocarcinoma . RIA .
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
JAR
Choriocarcinoma
RIA
PACAP38
Megjelenés:Journal of Molecular Neuroscience. - 42 : 3 (2010), p. 450-458. -
További szerzők:Boronkai A. Reglődi Dóra (Idegtudományok) Rácz B. (Pécs) Németh József (1954-) (vegyész, analitikus) Kiss Péter (Pécs) Lubics Andrea (Pécs) Tóth Gábor (Szeged) Horváth Gábor (Pécs) Varga T. (Pécs) Szogyi D. (Pécs) Fonagy E. Farkas J. (Pécs) Barakonyi A. Bellyei Szabolcs Szereday László Koppán Miklós Tamás A. (Pécs)
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12.

001-es BibID:BIBFORM080931
035-os BibID:(WoS)000483415800005 (Scopus)85071715733
Első szerző:Chistiakova, Marina
Cím:Distinct Heterosynaptic Plasticity in Fast Spiking and Non-Fast-Spiking Inhibitory Neurons in Rat Visual Cortex / Marina Chistiakova, Vladimir Ilin, Matvey Roshchin, Nicholas Bannon, Alexey Malyshev, Zoltán Kisvárday, Maxim Volgushev
Dátum:2019
ISSN:0270-6474 1529-2401
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
folyóiratcikk
Megjelenés:Journal Of Neuroscience. - 39 : 35 (2019), p. 6865-6878. -
További szerzők:Ilin, Vladimir Roshchin, Matvey Bannon, Nicholas Malyshev, Alexey Kisvárday Zoltán (1957-) (biológus, neurobiológus) Volgushev, Maxim
Pályázati támogatás:NAP-2-2017-1.2.1-NKP-00002
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