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1.

001-es BibID:BIBFORM057430
Első szerző:Auricchio, Renata
Cím:The frequency of coeliac disease (CD) in high-risk young children from families with CD : the Preventcd cohort / R. Auricchio, C. Hogen Esch, G. Castillejo, E. Mummert, E. Bravi, I. Korponay-Szabo, S. Koletzko, L. Greco, R. Troncone, M. L. Mearin, The PreventCD Study Group
Dátum:2011
Tárgyszavak:Orvostudományok Klinikai orvostudományok idézhető absztrakt
Megjelenés:Journal of Pediatric Gastroenterology and Nutrition 52 : Suppl. 2 (2011), p. E7. -
További szerzők:Hogen Esch, Caroline Castillejo, Gemma (gyermekgyógyász, gasztroenterológus) Mummert, Eckart Bravi, Enzo (biológus) Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Koletzko, Sibylle Greco, Luigi Troncone, Riccardo Mearin, Maria Luisa the PREVENTCD Study Group
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2.

001-es BibID:BIBFORM011928
Első szerző:Caja, Sergio
Cím:Inhibition of transglutaminase 2 enzymatic activity ameliorates the anti-angiogenic effects of coeliac disease autoantibodies / Caja S., Myrsky E., Korponay-Szabo I., Nadalutti C., Sulic A. M., Lavric M., Sblattero D., Marzari R., Collighan R., Mongeot A., Griffin M., Maki M., Kaukinen K., Lindfors K.
Dátum:2010
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Scandinavian Journal of Gastroenterology. - 45 : 4 (2010), p. 421-427. -
További szerzők:Myrsky, Essi Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Nadalutti, Cristina Sulic, Ana-Marija Lavric, Miha Sblattero, Daniele Marzari, Roberto Collighan, Russell Mongeot, Alexandre Griffin, Martin Mäki, Markku Kaukinen, Katri Lindfors, Katri
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3.

001-es BibID:BIBFORM044302
Első szerző:Collin, Pekka
Cím:Antiendomysial and antihuman recombinant tissue transglutaminase antibodies in the diagnosis of coeliac disease : a biopsy-proven European multicentre study / Collin Pekka, Kaukinen Katri, Vogelsang Harald, Korponay-Szabo Ilma, Sommer Rudolf, Schreier Elisabeth, Volta Umberto, Granito Alessandro, Veronesi Lorenza, Mascart Francoise, Ocmant Annick, Ivarsson Anneli, Lagerqvist Carina, Bürgin-Wolff Annemarie, Hadziselimovic Faruk, Furlano Raoul I., Sidler Marc A., Mulder Chris J. J., Goerres Marije S., Mearin M. Luisa, Ninaber Maarten K., Gudmand-Hoyer Eivind, Fabiani Elisabetta, Catassi Carlo, Tidlund Helena, Alainentalo Lisbeth, Maki Markku
Dátum:2005
ISSN:0954-691X
Megjegyzések:OBJECTIVE:To investigate the value of serum antitissue transglutaminase IgA antibodies (IgA-TTG) and IgA antiendomysial antibodies (IgA-EMA) in the diagnosis of coeliac disease in cohorts from different geographical areas in Europe. The setting allowed a further comparison between the antibody results and the conventional small-intestinal histology.METHODS:A total of 144 cases with coeliac disease [median age 19.5 years (range 0.9-81.4)], and 127 disease controls [median age 29.2 years (range 0.5-79.0)], were recruited, on the basis of biopsy, from 13 centres in nine countries. All biopsy specimens were re-evaluated and classified blindly a second time by two investigators. IgA-TTG were determined by ELISA with human recombinant antigen and IgA-EMA by an immunofluorescence test with human umbilical cord as antigen.RESULTS:The quality of the biopsy specimens was not acceptable in 29 (10.7%) of 271 cases and a reliable judgement could not be made, mainly due to poor orientation of the samples. The primary clinical diagnosis and the second classification of the biopsy specimens were divergent in nine cases, and one patient was initially enrolled in the wrong group. Thus, 126 coeliac patients and 106 controls, verified by biopsy, remained for final analysis. The sensitivity of IgA-TTG was 94% and IgA-EMA 89%, the specificity was 99% and 98%, respectively.CONCLUSIONS:Serum IgA-TTG measurement is effective and at least as good as IgA-EMA in the identification of coeliac disease. Due to a high percentage of poor histological specimens, the diagnosis of coeliac disease should not depend only on biopsy, but in addition the clinical picture and serology should be considered
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:European Journal Of Gastroenterology & Hepatology. - 17 : 1 (2005), p. 85-91. -
További szerzők:Kaukinen, Katri Vogelsang, Harald Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Sommer, Rudolf Schreier, Elisabeth Volta, Umberto Granito, Alessandro Veronesi, Lorenza Mascart, Francoise Ocmant, Annick Ivarsson, Anneli Lagerqvist, Carina Bürgin-Wolff, Annemarie Hadziselimovic, Faruk Furlano, Raoul I. Sidler, Marc A. Mulder, Chris J. Goerres, Marije S. Mearin, Maria Luisa Ninaber, Maarten K. Gudmand-Hoyer, Eivind Fabiani, Elisabetta Catassi, Carlo Tidlund, Helena Alainentalo, Lisbeth Mäki, Markku
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4.

001-es BibID:BIBFORM011939
Első szerző:Dahlbom, Ingrid
Cím:Prediction of clinical and mucosal severity of coeliac disease and dermatitis herpetiformis by quantification of IgA/IgG serum antibodies to tissue transglutaminase / Ingrid Dahlbom, Ilma R. Korponay-Szabó, Judit B. Kovács, Zsuzsanna Szalai, Markku Mäki, Tony Hansson
Dátum:2010
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal of Pediatric Gastroenterology and Nutrition. - 50 : 2 (2010), p. 140-146. -
További szerzők:Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Kovács Judit B. Szalai Zsuzsanna Mäki, Markku Hansson, Tony
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5.

001-es BibID:BIBFORM040844
Első szerző:Dezsőfi Antal
Cím:Frequencies of Genetic Polymorphisms of TLR4 and CD14 and of HLA-DQ Genotypes in Children With Celiac Disease, Type 1 Diabetes Mellitus, or Both / Dezsőfi, A., Szebeni, B., Hermann, CS., Kapitány, A., Veres, G., Sipka, S., Körner, A., Madácsy, L., Korponay-Szabó, I., Rajczy, K., Arató, A.
Dátum:2008
ISSN:0277-2116
Megjegyzések:OBJECTIVES: Besides the central role of the adaptive immune system, a disturbance of innate immunity is also involved in the pathogenesis of celiac disease (CD). Inasmuch as CD and type 1 diabetes mellitus (T1DM) frequently coexist because of a common genetic predisposition, our aim was to study the frequency of CD14 C-260T and TLR4 A+896G single nucleotide polymorphisms (SNPs) and the distribution of HLA-DQ genotypes in children affected by CD, T1DM, or both. PATIENTS AND METHODS: TLR4 and CD14 SNPs were tested by polymerase chain reaction, followed by restriction fragment length polymorphism analysis in 80 children with T1DM, 100 children with CD, and 47 children with both CD and T1DM. Determination of HLA-DQ alleles was done by sequence-specific polymerase chain reaction. Frequencies were compared with those of healthy control children. RESULTS: The prevalence of the homozygous CD14 C-260TT genotype was significantly (P = 0.0081) lower in children with T1DM but not in those with CD and T1DM, compared with control children. No difference was found in the genotype and allele frequencies of TLR4 between the studied groups. In patients with T1DM, the frequency of the homozygous HLA-DQ8 genotype was significantly higher than in CD, whereas the frequency of homozygous or heterozygous HLA-DQ2 genotypes did not differ from that in control children. In patients with CD, both homozygous and heterozygous HLA-DQ2 genotypes were significantly more frequent than in the control and T1DM groups, and no elevation in the frequency of the HLA-DQ8 genotypes was observed. In patients with T1DM and those with CD and T1DM, the occurrence of HLA-DQ2/8 heterozygosity was significantly higher than in children with CD only and in control children. CONCLUSIONS: Our results suggest that in patients with T1DM, the CD14 C-260TT homozygous genotype increases the risk for the development of CD. The distribution of HLA-DQ genotype is different in children with CD and T1DM than in children with CD or T1DM only. Determination of the HLA-DQ genotype in children with T1DM may help in estimating the risk for the development of CD.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal Of Pediatric Gastroenterology And Nutrition. - 47 : 3 (2008), p. 283-287. -
További szerzők:Szebeni Beáta Hermann, CS. Kapitány Anikó (1979-) (molekuláris biológus) Veres Gábor (1969-2020) (csecsemő- és gyermekgyógyász, gasztroenterológus) Sipka Sándor (1945-) (laboratóriumi szakorvos) Körner Anna Madácsy László (Szeged) Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Rajczy Katalin Arató András
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6.

001-es BibID:BIBFORM002290
Első szerző:Garcia-Horsman, J. Arturo
Cím:Deficient activity of mammalian prolyl oligopeptidase on the immunoactive peptide digestion in coeliac disease / J. Arturo Garcia-Horsman, Jarkko I. Venäläinen, Olli Lohi, I. Seppo Auriola, Ilma R. Korponay-Szabó, Katri Kaukinen, Markku Mäki, Pekka T. Männistö
Dátum:2007
Megjegyzések:Objective. Gliadin digestion-resistant peptides are harmful in coeliac disease (CD), and initiate an autoimmune reaction that cause a cascade of symptoms. The role of the endogenous prolyl oligopeptidase (POP) is still not clear, and its activity over gliadin immunoactive peptides has not been fully established. Our objective was therefore to determine the endogenous POP protein level, tissue distribution and total activity in normal and CD epithelia, to evaluate tissue peptidase activity over gliadin peptides, and compare this with activities of mammalian POP and rat intestinal extracts. Material and methods. POP was assayed in biopsy preparations enzymatically and by Western blot analysis. Distribution was studied by immunohistochemistry using a specific POP antibody. Peptide cleavage was followed by mass spectroscopy-highperformance liquid chromatography (MS-HPLC). Results. There was no difference in POP activity between normal and CD samples, but those from active CD subjects had an even higher ability to degrade the 33-mer peptide than those from treated CD and healthy humans. POP locates intracellularly in epithelia, similarly to dipeptidyl peptidase IV (DPPIV), but the latter is clearly found in normal microvilli but less so in diseased microvilli. Mammalian POP is unable to digest 33-mer peptide, which, conversely, is a POP inhibitor. Rat intestine is more effective than human intestine in cleaving the 33-mer peptide. However, the products are still harmful epitopes. A surplus of POP eliminates 12-mer and 19-mer peptide products. Conclusions. The results rule out a causative role of POP in the pathogenesis of CD and strongly suggest that other peptidases are needed to eliminate gliadin-derived, immunoactive and toxic peptides larger than 33-mer, which is a POP inhibitor.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
coeliac disease
digestion
gliadin
peptidase
prolyl oligopeptidase
Megjelenés:Scandinavian Journal of Gastroenterology 42 : 5 (2007), p. 562-571. -
További szerzők:Venäläinen, Jarkko I. Lohi, Olli Auriola, I. Seppo Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Kaukinen, Katri Mäki, Markku Männistö, Pekka T.
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7.

001-es BibID:BIBFORM044267
Első szerző:Giersiepen, Klaus
Cím:Accuracy of Diagnostic Antibody Tests for Coeliac Disease in Children / Giersiepen Klaus, Lelgemann Monika, Stuhldreher Nina, Ronfani Luca, Husby Steffen, Koletzko Sibylle, Korponay-Szabó Ilma R., the ESPGHAN Working Group on Coeliac Disease Diagnosis
Dátum:2012
ISSN:0277-2116
Megjegyzések:OBJECTIVE: The aim of this study was to summarise the evidence from 2004 toSeptember 2009 on the performance of laboratory-based serological and point ofcare (POC) tests for diagnosing coeliac disease (CD) in children using histology as reference standard.PATIENTS AND METHODS: We searched MEDLINE and EMBASE for studies reporting onchildren for tests based on IgA and IgG anti-gliadin (AGA), endomysial (EmA),anti-transglutaminase-2 (TG2), and anti-deamidated gliadin peptides (DGP)antibodies or POC tests. For inclusion, histological analysis of duodenalbiopsies and sensitivity and specificity for index tests had to be reported. Datawere pooled and summary measures calculated for sensitivity, specificity,positive and negative likelihood ratios ("LR+", "LR-"), and diagnostic oddsratios (DOR). In case of elevated statistical heterogeneity, studies reaching 90%sensitivity or specificity were reported.RESULTS: A total of 2510 articles were reviewed; 16 entered meta-analysis,reporting on 3110 patients (1876 with CD, 1234 without CD). For IgA-EmA,sensitivity was ?90% in 7/11 studies and pooled specificity 98.2%. ForIgA-anti-TG2, 11/15 studies yielded sensitivities ?90% and 13/15 specificities?90%. For IgA-DGP, sensitivity ranged between 80.7% and 95.1% (specificity86.3%-93.1%); for IgG-DGP between 80.1% and 98.6% (specificity 86.0-96.9%).IgA-EmA had the highest pooled DOR (554) and LR+ (31.8) for a laboratory test,followed by IgA-anti-TG2, IgG-DGP, IgA-DGP and IgA-AGA. POC tests showed a pooledsensitivity of 96.4% for IgA-TG2 (specificity 97.7%).CONCLUSIONS: IgA-EmA and IgA-anti-TG2 tests appear highly accurate to diagnoseCD. IgG-anti-DGP tests may help in excluding CD. IgA-AGA and IgA-DGP tests showinferior accuracy. POC tests may achieve high accuracy in the hands ofexperienced readers, but IgA-anti-TG2/EmA were superior.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal Of Pediatric Gastroenterology And Nutrition. - 54 : 2 (2012), p. 229-241. -
További szerzők:Lelgemann, Monika Stuhldreher, Nina Ronfani, Luca Husby, Steffen Koletzko, Sibylle Korponay-Szabó Ilma (1959-) (gyermekgyógyász) the ESPGHAN Working Group on Coeliac Disease Diagnosis
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8.

001-es BibID:BIBFORM057434
Első szerző:Gyimesi Judit
Cím:Coeliac disease manifestation in early age in a prospectively followed contemporary natural history cohort of first degree relatives / J. Gyimesi, E. Nemes, E. Vajda-Nagy, O. Kadenczki, I. Korponay-Szabo, PreventCD Study Group
Dátum:2013
Tárgyszavak:Orvostudományok Klinikai orvostudományok idézhető absztrakt
Megjelenés:United European Gastroenterology Journal 1 : Suppl. 1 (2013), p. A581. -
További szerzők:Nemes Éva (1957-) (csecsemő- és gyermekgyógyász, gasztroenterológus) Vajda-Nagy Erika (dietetikus) Kadenczki Orsolya (1974-) (csecsemő- és gyermekgyógyász) Korponay-Szabó Ilma (1959-) (gyermekgyógyász) PreventCD Group
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9.

001-es BibID:BIBFORM091976
Első szerző:Husby, Steffen
Cím:European Society Paediatric Gastroenterology, Hepatology and Nutrition Guidelines for Diagnosing Coeliac Disease 2020 / Steffen Husby, Sibylle Koletzko, Ilma Korponay-Szabó, Kalle Kurppa, Maria Luisa Mearin, Carmen Ribes-Koninckx, Raanan Shamir, Riccardo Troncone, Renata Auricchio, Gemma Castillejo, Robin Christensen, Jernej Dolinsek, Peter Gillett, Asbjørn Hróbjartsson, Tunde Koltai, Markku Maki, Sabrina Mai Nielsen, Alina Popp, Ketil Størdal, Katharina Werkstetter, Margreet Wessels
Dátum:2020
ISSN:0277-2116
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
folyóiratcikk
Megjelenés:Journal of pediatric gastroenterology and nutrition. - 70 : 1 (2020), p. 141-156. -
További szerzők:Koletzko, Sibylle Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Kurppa, Kalle Mearin, Maria Luisa Ribes-Koninckx, Carmen Shamir, Raanan (gyermekgyógyász) Troncone, Riccardo Auricchio, Renata Castillejo, Gemma (gyermekgyógyász, gasztroenterológus) Christensen, Robin Dolinśek, Jernej Gillett, Peter Hróbjartsson, Asbjørn Koltai Tünde Mäki, Markku Nielsen, Sabrina Mai Popp, Alina Størdal, Ketil Werkstetter, Katharina (gyermekgyógyász, gasztroenterológus) Wessels, Margreet
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10.

001-es BibID:BIBFORM044266
Első szerző:Husby, Steffen
Cím:European Society for Pediatric Gastroenterology, Hepatology, and Nutrition Guidelines for the Diagnosis of Coeliac Disease / Husby, S., Koletzko, S., Korponay-Szabó, I. R., Mearin, M. L., Phillips, A., Shamir, R., Troncone, R., Giersiepen, K., Branski, D., Catassi, C., Lelgeman, M., Mäki, M., Ribes-Koninckx, C., Ventura, A., Zimmer, K. P., the ESPGHAN Working Group on Coeliac Disease Diagnosis, the ESPGHAN Gastroenterology Committee
Dátum:2012
ISSN:0277-2116
Megjegyzések:OBJECTIVE: Diagnostic criteria for coeliac disease (CD) from the European Society for Paediatric Gastroenterology, Hepatology, and Nutrition (ESPGHAN) were published in 1990. Since then, the autoantigen in CD, tissue transglutaminase, has been identified; the perception of CD has changed from that of a rather uncommon enteropathy to a common multiorgan disease strongly dependent on the haplotypes human leukocyte antigen (HLA)-DQ2 and HLA-DQ8; and CD-specific antibody tests have improved.METHODS:A panel of 17 experts defined CD and developed new diagnostic criteria based on the Delphi process. Two groups of patients were defined with different diagnostic approaches to diagnose CD: children with symptoms suggestive of CD (group 1) and asymptomatic children at increased risk for CD (group 2). The 2004 National Institutes of Health/Agency for Healthcare Research and Quality report and a systematic literature search on antibody tests for CD in paediatric patients covering the years 2004 to 2009 was the basis for the evidence-based recommendations on CD-specific antibody testing.RESULTS:In group 1, the diagnosis of CD is based on symptoms, positive serology, and histology that is consistent with CD. If immunoglobulin A anti-tissue transglutaminase type 2 antibody titers are high (>10 times the upper limit of normal), then the option is to diagnose CD without duodenal biopsies by applying a strict protocol with further laboratory tests. In group 2, the diagnosis of CD is based on positive serology and histology. HLA-DQ2 and HLA-DQ8 testing is valuable because CD is unlikely if both haplotypes are negative.CONCLUSIONS:The aim of the new guidelines was to achieve a high diagnostic accuracy and to reduce the burden for patients and their families. The performance of these guidelines in clinical practice should be evaluated prospectively.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
celiac disease
Megjelenés:Journal Of Pediatric Gastroenterology And Nutrition 54 : 1 (2012), p. 136-160. -
További szerzők:Koletzko, Sibylle Korponay-Szabó Ilma (1959-) (gyermekgyógyász) Mearin, Maria Luisa Phillips, A. Shamir, R. Troncone, Riccardo Giersiepen, Klaus Branski, D. Catassi, Carlo Lelgemann, Monika Mäki, Markku Ribes-Koninckx, Carmen Ventura, Alessandro Zimmer, Klaus-Peter the ESPGHAN Working Group on Coeliac Disease Diagnosis the ESPGHAN Gastroenterology Committee
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11.

001-es BibID:BIBFORM044301
Első szerző:Kaukinen, Katri
Cím:Small-bowel mucosal transglutaminase 2-specific IgA deposits in coeliac disease without villous atrophy : a prospective and randomized clinical study / Kaukinen Katri, Peräaho Markku, Collin Pekka, Partanen Jukka, Woolley Nina, Kaartinen Tanja, Nuutinen Tuula, Halttunen Tuula, Mäki Markku, Korponay-Szabo Ilma
Dátum:2005
ISSN:0036-5521
Megjegyzések:OBJECTIVE:In coeliac disease, autoantibodies directed against transglutaminase 2 are produced in small-bowel mucosa, and they have been found to be deposited extracellularly. The aim of this study was to investigate whether such mucosal IgA deposits are important in the diagnostic work-up of early-stage coeliac disease without small-bowel mucosal villous atrophy.MATERIAL AND METHODS:Forty-one adults suspected of coeliac disease owing to increased density of mucosal gamma(delta)+ intraepithelial lymphocytes but normal villous morphology were randomized to gluten challenge or a gluten-free diet for 6 months. Clinically and histologically verified gluten dependency was compared with existence of small-bowel mucosal transglutaminase 2-specific extracellular IgA deposits and (coeliac disease-type) HLA DQ2 and DQ8; 34 non-coeliac subjects and 18 patients with classical coeliac disease served as controls.RESULTS:Of the 41 patients, 5 in the challenge group and 6 in the gluten-free diet group were clinically gluten sensitive; all 11 had HLA DQ2 or DQ8. Ten of these 11 patients showed transglutaminase 2-targeted mucosal IgA deposits, which were dependent on gluten consumption. Minimal IgA deposits were seen in only 3 out of 30 patients with suspected coeliac disease without any clinically detected gluten dependency. The deposits were found in all classical coeliac patients and in none of the non-coeliac control subjects.CONCLUSIONS:Clinically pertinent coeliac disease exists despite normal small-bowel mucosal villous architecture. Mucosal transglutaminase 2-specific IgA deposits can be utilized in detecting such patients with genetic gluten intolerance
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Scandinavian Journal Of Gastroenterology. - 40 : 5 (2005), p. 564-572. -
További szerzők:Peräaho, Markku Collin, Pekka Partanen, Jukka Woolley, Nina Kaartinen, Tanja Nuutinen, Tuula Halttunen, Tuula Mäki, Markku Korponay-Szabó Ilma (1959-) (gyermekgyógyász)
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12.

001-es BibID:BIBFORM086069
Első szerző:Korponay-Szabó Ilma (gyermekgyógyász)
Cím:Is coeliac disease a dominantly inherited disorder? / Korponay-Szabó Ilma Rita, Kapitány Anikó, Kovács J. B., Lőrincz M., Opre J., Nemes Éva., Tumpek J., Sipka S.
Dátum:2005
ISSN:0277-2116
Tárgyszavak:Orvostudományok Klinikai orvostudományok idézhető absztrakt
folyóiratcikk
Megjelenés:Journal of Pediatric Gastroenterology and Nutrition. - 40 : 5 (2005), p. 638. -
További szerzők:Kapitány Anikó (1979-) (molekuláris biológus) Kovács J. Béla Lőrincz Margit Opre Judit (Kenézy Gyula Kórház) Nemes Éva (1957-) (csecsemő- és gyermekgyógyász, gasztroenterológus) Tumpek Judit (1944-) (orvosi laboratóriumi szakorvos) Sipka Sándor (1945-) (laboratóriumi szakorvos)
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