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1.

001-es BibID:BIBFORM030625
Első szerző:Bodnár Ibolya
Cím:Effect of neonatal treatment with monosodium glutamate on dopaminergic and L-DOPA-ergic neurons of the medial basal hypothalamus and on prolactin and MSH secretion of rats / Ibolya Bodnár, Pál Göőz, Hitoshi Okamura, Béla E. Tóth, Miklós Vecsernyés, Béla Halász, György M. Nagy
Dátum:2001
ISSN:0361-9230
Megjegyzések:The effect of neonatal treatment with monosodium L-glutamate (MSG) on the dopaminergic systems of the medial basal hypothalamus has been investigated using tyrosine hydroxylase (TH) and aromatic L-amino acid decarboxylase (AADC) immunocytochemistry. Changes in plasma levels of prolactin (PRL) and alpha-melanocyte-stimulating hormone (MSH) have also been determined in intact and in MSG-treated rats after inhibition of TH by alpha-methyl-p-tyrosine (alpha-MpT) or without inhibition of enzyme activity. Monosodium glutamate resulted in a 40% reduction in the number of TH immunopositive tuberoinfundibular neurons, but no change in the number of AADC-positive tuberoinfundibular nerve cells, indicating that this reduction has occurred mainly in TH-positive but AADC-negative elements, i.e., in L-DOPA-ergic neurons. In contrast, MSG did not cause changes in the number of TH and AADC immunoreactive neurons of the periventriculohypophysial and tuberohypophysial dopaminergic systems, and it did not influence basal plasma PRL levels. alpha-methyl-p-tyrosine has increased plasma PRL concentrations in both control and MSG-treated rats of both sexes, but significantly higher responses were detected in females. None of the treatments had any effect on plasma MSH level. These findings suggest that MSG affects primarily L-DOPA-ergic neurons located in the ventrolateral part of the arcuate nucleus, but not dopaminergic neurons situated in the dorsomedial part of the arcuate nucleus; neither PRL nor MSH secretion is altered by MSG; a significant sex difference exists in the pituitary PRL response to inhibition of TH, and this response is not affected by MSG.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
arcuate nucleus
tyrosine hydroxylase
aromatic L-amino acid decarboxylase
immunocytochemistry
Megjelenés:Brain Research Bulletin 55 : 6 (2001), p. 767-774. -
További szerzők:Göőz Pál Okamura, Hitoshi Tóth E. Béla (1960-) (orvos) Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Halász Béla Nagy György M.
Pályázati támogatás:T-30748
OTKA
T-029004
OTKA
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2.

001-es BibID:BIBFORM030573
035-os BibID:PMID:9739113 WOS:000076241100010
Első szerző:DeMaria, Jamie E.
Cím:The effect of neurointermediate lobe denervation on hypothalamic neuroendocrine dopaminergic neurons / Jamie E. DeMaria, Dora Zelena, Miklós Vecsernyés, György M. Nagy, Marc E. Freeman
Dátum:1998
ISSN:0006-8993
Megjegyzések:The contribution of tuberohypophyseal and periventricular-hypophyseal dopaminergic neurons to the regulation of the secretion of prolactin (PRL) has yet to be clarified. In this study, we used pituitary stalk compression to disrupt hypothalamic neural input to the neurointermediate lobe (NIL). Neurointermediate lobe denervation (NIL-D) selectively disrupts the axons of tuberohypophyseal and periventricular-hypophyseal dopaminergic neurons, while leaving tuberoinfundibular dopaminergic neurons and the vascular supply of the pituitary gland intact. NIL-D was performed in ovariectomized (OVX) rats. The concentration of DA and 3,4-dihydroxyphenylacetic acid (DOPAC) in the median eminence (ME) and various regions of the pituitary gland of OVX and OVX+NIL-D rats were measured by HPLC-EC. The concentration of PRL, alpha-melanocyte stimulating hormone (alpha-MSH), and luteinizing hormone (LH) in serum were determined by radioimmunoassay. Successful NIL-D was confirmed by increased water intake. One week after NIL-D, serum PRL and alpha-MSH were elevated, but there was no change in the concentration of LH in serum. The concentration of DA was increased in the median eminence (ME), decreased in the outer zone of the anterior lobe (AL-OZ), as well as the intermediate (IL) and neural lobes (NL), and remained unchanged in the inner zone of the anterior lobe (AL-IZ). The concentration of DOPAC was increased in the ME and NL, decreased in the IL, and remained unchanged in both the AL-IZ and AL-OZ. These data confirm that pituitary stalk compression denervates the NIL. Moreover, decreases in the concentration of DA in the IL and AL-OZ, coupled with elevation of serum PRL and alpha-MSH indicate that DA from the NIL contributes to the increased inhibition of the secretion of PRL and alpha-MSH in OVX rats.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
dopamine
DOPAC
neurointermediate lobe denervation
prolactin
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 806 : 1 (1998), p. 89-94. -
További szerzők:Zelena Dóra Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Nagy György M. Freeman, Marc E.
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3.

001-es BibID:BIBFORM013978
Első szerző:Fehér Pálma (gyógyszerész)
Cím:Dephosphorylation/inactivation of tyrosine hydroxylase at the median eminence of the hypothalamus is required for suckling-induced prolactin and adrenocorticotrop hormone responses / Fehér Pálma, Oláh Márk, Bodnár Ibolya, Hechtl Dániel, Bácskay Ildikó, Juhász Béla, Nagy György M., Vecsernyés Miklós
Dátum:2010
ISSN:0361-9230
Megjegyzések:Wehave recently found that dopamine (DA) released from terminals of the hypothalamic neuroendocrine dopamine (NEDA) neurons plays a role not only in prolactin (PRL), but also in adrenocorticotrop hormone (ACTH) secretion, without having any influence on alfa-melanocyte-stimulating hormone (alfa-MSH) release in lactating dams. The aim of our present studies was to further investigate this DAerg regulation of ACTH using consecutively applied physiological stimulation (suckling) and pharmacological inhibition of the rate-limiting enzyme of DA synthesis (tyrosine hydroxylase, TH) by alfa-methyl-p-tyrosine (alfa-MpT) that acutely affect secretion of these pituitary hormones during lactation. Following 4 h separation period, two experimental groups were formed. In the first group, lactating rats were assembled with their litters for 60 min prior to alfa-MpT. In the second group, the alfa-MpT was injected first and 60 min later suckling stimulus was applied. Plasma samples were taken in every 15 min during the 90 min experimental period. Concentrations of plasma PRL, ACTH and alfa-MSH were measured by specific RIAs. Both stimuli applied in the first sequence, significantly elevated plasma PRL and ACTH levels in separated lactating dams, without having any effect on alfa-MSH secretion. Suckling applied in the first sequence was able to block the alfa-MpT-induced elevation of ACTH secretion, while PRL response was also significantly attenuated. alfa-MpT pretreatment prevented both PRL and ACTH responses to suckling stimulus. Investigating the dephosphorylation/inactivation of TH in the arcuate nucleus-ME (TIDA) regions, no pTH-immunoreactive perikarya or terminals can be found in continuously suckled dams. In contrast, after 4 h separation of the mothers from their litters, pTH-immunoreactivity can be clearly visualized in the external zone of ME. In alfa-MpT pretreated mothers following 4 h separation no pTH positive terminals are visible. No changes in the TH immunostaining can be observed in any of these experimental groups. In conclusion, dephosphorylation/inactivation of TH (the rate-limiting enzyme of the DA biosynthesis) in NEDA neurons is required for suckling-induced PRL and ACTH responses.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
prolactin
Megjelenés:Brain Research Bulletin. - 82 : 1-2 (2010), p. 141-145. -
További szerzők:Oláh Márk Bodnár Ibolya Hechtl Dániel Bácskay Ildikó (1969-) (gyógyszerész, gyógyszertechnológus) Juhász Béla (1978-) (kísérletes farmakológus) Nagy György M. Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus)
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4.

001-es BibID:BIBFORM030669
Első szerző:Horváth Katalin M.
Cím:Suckling-induced increase in cyclic AMP exclusively in the central region of the rat adenohypophysis / Katalin M. Horváth, Anikó Somogyvári-Vigh, Zoltán Kandár, Miklós Vecsernyés, Akira Arimura, György M. Nagy
Dátum:2000
ISSN:0006-8993
Megjegyzések:Investigating the cellular events in the pituitary gland, the intracellular cyclic AMP (cAMP) of the neural lobe (NL), intermediate lobe (IL), the inner (IZ-AL) and outer zone (OZ-AL) of the anterior lobe (AL) have been measured during the suckling stimulus. Ten-minutes suckling, parallel to the elevation of plasma PRL, induced a significant increase of cAMP concentration in the IZ-AL. In contrast, 10- and 30-min suckling resulted in a decrease of cAMP level in the NL. Changes in cAMP of the OZ-AL and the IL as well as in the plasma level of alpha-MSH could not be detected. These region-specific changes of cAMP in the pituitary gland during suckling stimulus seems to be related to interacting neuroendocrine signals delivered concomitantly from the hypothalamus and from the NIL to the IZ-AL.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
suckling stimulus
inner zone of adenohypophysis
prolactin
cyclic AMP
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 873 : 1 (2000), p. 140-144. -
További szerzők:Somogyvári-Vigh Anikó Kandár Zoltán Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Arimura, Akira Nagy György M.
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5.

001-es BibID:BIBFORM030576
Első szerző:Julesz János
Cím:Central effects of tricyclic compounds on the endocrine system : an in vitro study / J. Julesz, M. Gálfi, J. Molnár, M. Vecsernyés
Dátum:1992
ISSN:0079-6123
Megjegyzések:The present study involves the effects on corticotropin (ACTH) release of neuro- and thymoleptic tricyclic antidepressant compounds (TrcACs: chlorpromazine, promethazine, haloperidol, imipramine, amitriptyline) and their interactions with lysine-8-vasopressin (LVP) and corticosterone (B). As an in vitro model, 14-day monolayer pituitary cell cultures of Wistar rats were employed. The ACTH concentrations of the supernatant media were measured by radioimmunoassay. TrcACs augmented ACTH release; their combination with LVP, however, did not result in further stimulation; moreover, when combined with TrcACs + LVP, B did not inhibit, but rather paradoxically increased their ACTH-releasing action. As none of these phenomena were followed by relevant changes in intracellular cyclic adenosine monophosphate content, the mechanism of action may be proposed to involve a protein kinase C route.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
egyetemen (Magyarországon) készült közlemény
Megjelenés:Progress In Brain Research. - 91 (1992), p. 89-92. -
További szerzők:Gálfi Márta Molnár J. Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus)
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6.

001-es BibID:BIBFORM030636
Első szerző:Kovács Gábor L. (Szeged)
Cím:Limbic oxytocin and arginine 8-vasopressin in morphine tolerance and dependence / G. L. Kovács, F. Laczi, M. Vecsernyés, K. Hódi, G. Telegdy, F. A. László
Dátum:1987
ISSN:0014-4819
Megjegyzések:Immunoreactive oxytocin (OXT) and arginine8-vasopressin (AVP) levels were measured in limbic areas of the mouse brain (hippocampus, amygdala and basal forebrain). Peptides were measured by radioimmunoassay (RIA). Acute morphine treatment caused a naloxone-reversible increase in OXT content in all three brain regions. The AVP contents of the same brain areas, on the other hand, were not affected by acute morphine treatment. In mice rendered tolerant to/dependent on morphine with subcutaneous morphine pellets, the OXT levels in the limbic brain structures were in the control range (basal forebrain and amygdala) or even decreased (hippocampus). In the latter brain structure of the tolerant animals, the AVP content was also decreased. Naloxone-precipitated withdrawal syndrome in the tolerant/dependent animals resulted in abrupt increases in the OXT and AVP levels of the hippocampus and in the OXT content of the basal forebrain structures.
Tárgyszavak:Orvostudományok Gyógyszerészeti tudományok idegen nyelvű folyóiratközlemény külföldi lapban
morphine tolerance
morphine dependence
limbic brain regions
egyetemen (Magyarországon) készült közlemény
Megjelenés:Experimental Brain Research 65 : 2 (1987), p. 307-311. -
További szerzők:Laczi Ferenc (Szeged) Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Hódi Klára Telegdy Gyula (Szeged) László Ferenc A.
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7.

001-es BibID:BIBFORM030549
Első szerző:Kovács L. Gábor
Cím:Acute morphine treatment and morphine tolerance/dependence alter immunoreactive oxytocin levels in the mouse hippocampus / Gábor L. Kovács, Miklós Vecsernyés, Ferenc Laczi, Mária Faludi, Gyula Telegdy, Ferenc A. László
Dátum:1985
ISSN:0006-8993
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
hippocampal oxytocin
morphine
tolerance
withdrawal
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 328 : 1 (1985), p. 158-160. -
További szerzők:Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Laczi Ferenc (Szeged) Faludi Mária Telegdy Gyula (Szeged) László Ferenc A.
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8.

001-es BibID:BIBFORM030626
Első szerző:Laczi Ferenc (Szeged)
Cím:Effects of beta-endorphin2-9 on arginine-8-vasopressin and oxytocin levels in hypothalamic and limbic brain regions / Ferenc Laczi, Miklós Vecsernyés, Gábor L. Kovács, Gyula Szabó, Tamás Janáky, Gyula Telegdy, Ferenc A. László
Dátum:1987
ISSN:0006-8993
Megjegyzések:mmunoreactive arginine-8-vasopressin (AVP) and oxytocin (OXT) were measured in rat hypothalamic and limbic brain regions after the intracerebroventricular administration of beta-endorphin fragment 2-9 (beta E2-9). The peptide decreased the AVP content of the hippocampus and the OXT levels in the septum and amygdala. The present data favor the view that beta E2-9 interacts with limbic AVP- and OXT-systems.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
beta-endorphin2-9
arginine-8-vasopressin
oxytocin
brain area
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 403 : 1 (1987), p. 155-157. -
További szerzők:Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Kovács Gábor L. (Szeged) Szabó Gyula (Szeged) Janáky Tamás Telegdy Gyula (Szeged) László Ferenc A.
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9.

001-es BibID:BIBFORM030660
Első szerző:Radnai Balázs
Cím:Salsolinol induces a decrease in cyclic AMP at the median eminence and an increase at the adenohypophysis in lactating rats / Balázs Radnai, Zoltán Kandár, Anikó Somogyvári-Vigh, Zsuzsanna Mergl, Márk Oláh, Ferenc Fülöp, Miklós Vecsernyés, György M. Nagy
Dátum:2005
ISSN:0361-9230
Megjegyzések:Investigating the cellular events in the pituitary gland, the intracellular cyclic AMP (cAMP) of the median eminence (ME), neuro-intermediate lobe (NIL) and the anterior lobe (AL) have been measured following 15-min of intravenous injection of salsolinol (SAL). Parallel to the elevation of plasma prolactin (PRL), SAL induced a significant decrease of cAMP concentration in the ME. In contrast, SAL injection resulted in a significant increase of cAMP at the level of the AL. Changes in cAMP of the NIL as well as in the plasma level of vasopressin (VP) could not be detected. The observed changes in the level of cAMP following the acute treatment of SAL in the ME and the AL seems to be related to interacting neuroendocrine signals delivered from the ME to the AL through the long portal vessels to release PRL.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
pituitary gland
prolactin
tetrahydroisoquinoline
vasopressin
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research Bulletin 65 : 2 (2005), p. 105-110. -
További szerzők:Kandár Zoltán Somogyvári-Vigh Anikó Mergl Zsuzsanna Oláh Márk Fülöp Ferenc (Szeged) Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Nagy György M.
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10.

001-es BibID:BIBFORM030563
Első szerző:Sarnyai Zoltán
Cím:Brain corticotropin-releasing factor mediates 'anxiety-like' behavior induced by cocaine withdrawal in rats / Zoltán Sarnyai, Éva Bíró, János Gardi, Miklós Vecsernyés, János Julesz, Gyula Telegdy
Dátum:1995
ISSN:0006-8993
Megjegyzések:Anxiety is a key symptom of the cocaine withdrawal syndrome in human addicts, and it is considered to be one of the major factors in precipitating relapse to chronic cocaine abuse. Corticotropin-releasing factor (CRF) plays an important role in the pathophysiology of anxiety and depression, and it may also be involved in the acute behavioral and neuroendocrine actions of cocaine. The role of endogenous CRF in cocaine withdrawal-induced anxiety was investigated in the present study. Animals were subjected to chronic cocaine (20 mg/kg, intraperitoneally, once a day for 14 days) administration. Rats tested 30 min after the last cocaine injection did not show withdrawal anxiety on the elevated plus maze or any alterations in brain CRF levels. Withdrawal (48 h) from chronic cocaine administration produced an intense anxiety-like behavior characterized by decreased open arm exploration. Immunoreactive CRF (CRF-LI) levels were selectively altered in the hypothalamus, in the amygdala and in the basal forebrain structures at the time of the behavioral anxiety, reflecting an increased activity of brain CRF systems. Daily intracerebroventricular (i.c.v.) pretreatment with an immunoserum raised against CRF completely prevented the development of anxiety induced by cocaine withdrawal. These data suggest that extrahypothalamic-limbic CRF hypersecretion may be involved in the development of anxiety related to cocaine withdrawal and that the CRF system may be a useful target for new pharmacotherapies for cocaine withdrawal and relapse.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
cocaine withdrawal
corticotropin-releasing factor
anxiety
plus maze
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 675 : 1-2 (1995), p. 89-97. -
További szerzők:Bíró Éva Gardi János Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Julesz János Telegdy Gyula (Szeged)
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11.

001-es BibID:BIBFORM030557
Első szerző:Sarnyai Zoltán
Cím:Alterations of corticotropin-releasing factor-like immunoreactivity in different brain regions after acute cocaine administration in rats / Zoltán Sarnyai, Éva Bíró, János Gardi, Miklós Vecsernyés, János Julesz, Gyula Telegdy
Dátum:1993
ISSN:0006-8993
Megjegyzések:Corticotropin-releasing factor (CRF) may mediate some of the neuroendocrine and behavioral responses to cocaine. In this study, the distribution of CRF-like immunoreactivity (CRF-LI) was determined in the hypothalamus and in several extrahypothalamic brain regions after acute cocaine administration in handled rats. CRF-LI decreased dose-dependently with cocaine administration in the hypothalamus and in the basal-forebrain structures. A small dose of cocaine (7.5 mg/kg) decreased CRF-LI in the hippocampus and in the frontal cortex. A significant, selective, dose-dependent increase in CRF-LI was found in the amygdala after cocaine injection. None of the investigated doses of cocaine altered CRF-LI in the striatum. These results suggest that acute cocaine administration alters brain CRF systems to contribute behavioral and neuroendocrine responses to cocaine.
Tárgyszavak:Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
cocaine
corticotropin-releasing factor
hypothalamus
extrahypothalamic-limbic brain region
radioimmunoassay
egyetemen (Magyarországon) készült közlemény
Megjelenés:Brain Research 616 : 1-2 (1993), p. 315-319. -
További szerzők:Bíró Éva Gardi János Vecsernyés Miklós (1959-) (gyógyszertechnológus, endokrinológus) Julesz János Telegdy Gyula (Szeged)
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12.

001-es BibID:BIBFORM031998
035-os BibID:PMID:9227842 WOS:A1997XH42600010
Első szerző:Vecsernyés Miklós (gyógyszertechnológus, endokrinológus)
Cím:Effect of posterior pituitary denervation (PPD) on prolactin (PRL) and alpha-melanocyte-stimulating hormone (alpha-MSH) secretion of lactating rats / Miklós Vecsernyes, Krisztina Krempels, Béla E. Toth, János Julesz, Gábor B. Makara, György M. Nagy
Dátum:1997
Megjegyzések:Previous data have clearly suggested that the posterior pituitary (PP), consisting of neural lobe (NL) and intermediate lobe (IL), has a role in the control of anterior pituitary PRL secretion. However, basic aspects of this regulatory mechanism like (1), the role of an intact hypothalamic innervation of the PP as well as (2) the site of production of previously found PRL releasing substance(s) have not yet been characterized. Denervation of the PP (PPD) is an effective method for having a selective lesion of the innervation of PP, indeed, PPD results in a disappearance of neurosecretory materials from NL and tyrosine hydroxylase (TH) immunoreactivity from IL, leaving blood supply of all three lobes intact. Blood samples were taken from freely moving sham an PP-denervated lactating rats before and after 4-h separation from their pups and during the suckling stimulus. PPD blocks separation-induced depletion but only attenuates suckling induced release of PRL. Furthermore, it doubles plasma level of alpha-MSH during the entire sampling period, which has been used as a marker for in vivo secretory activity of IL cells. Lack of the separation-induced depression in plasma PRL of PPD animals can be partially restored by normalizing the diabetes insipidus with treatment of a vasopressin analogue, 1-desamino-8-D-arginine-vasopressin (dDAVP). In contrast, dDAVP, neither alone nor in combination with oxytocin (OXY), can change PPD-induced elevation of plasma alpha-MSH as well as attenuation of PRL response induced by suckling. It is concluded that: (1) contribution of the THDA system parallel to the confirmed role in the regulation of alpha-MSH seems to be crucial for the depletion of plasma PRL induced by separation but not for the elevation due to suckling stimulus, (2) intact hypothalamic innervations of both NL and IL, regulating water intake and the secretion of alpha-MSH, respectively, are necessary for normal secretory responses of AL during lactation, (3) as well as for the presence of PRF activity in PP, (4) which does not solely responsible for suckling-induced PRL release. Therefore, an interplay between several substances produced by NIL of the pituitary gland must have been responsible for the intact regulation of PRL secretion during lactation.
Tárgyszavak:Orvostudományok Gyógyszerészeti tudományok idegen nyelvű folyóiratközlemény külföldi lapban
egyetemen (Magyarországon) készült közlemény
Posterior pituitary denervation
Prolactin
a-MSH
Lactating rats
Megjelenés:Brain Research Bulletin 43 : 3 (1997), p. 313-319. -
További szerzők:Krempels Krisztina Tóth E. Béla (1960-) (orvos) Julesz János Makara Gábor B. Nagy György M.
Pályázati támogatás:13055
OTKA
20916
OTKA
2922
OTKA
16827
OTKA
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