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1.

001-es BibID:BIBFORM042418
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:A cerebrovascularis reaktivitás vizsgálata inzulin dependens diabetes mellitusban acetazolamid-teszttel / Fülesdi, B., Limburg, M., Neuwirth, Gy., Molnár, Cs., Valikovics, A., Csiba, L.
Dátum:1996
Tárgyszavak:Orvostudományok Klinikai orvostudományok magyar nyelvű folyóiratközlemény hazai lapban
Megjelenés:Orvosi Hetilap. - 137 : 39 (1996), p. 2137-2140. -
További szerzők:Limburg, Martien Neuwirth Gyula Molnár Csilla (1962-) (aneszteziológus) Valikovics Attila Csiba László (1952-) (neurológus, pszichiáter)
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2.

001-es BibID:BIBFORM042415
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Az acetazolamid szerepe a cerebralis hemodinamika vizsgálatában / Fülesdi, B., Limburg, M., Valikovics, A., Csiba, L.
Dátum:1995
Tárgyszavak:Orvostudományok Klinikai orvostudományok magyar nyelvű folyóiratközlemény hazai lapban
Megjelenés:Ideggyógyászati Szemle. - 48 : 5-6 (1995), p. 148-155. -
További szerzők:Limburg, Martien Valikovics Attila Csiba László (1952-) (neurológus, pszichiáter)
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3.

001-es BibID:BIBFORM042400
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:No relationship between cerebral blood flow velocity and cerebrovascular reserve capacity and contemporaneously measured glucose and insulin concentrations in diabetes mellitus / Fülesdi, B., Limburg, M., Bereczki, D., Molnár, C., Michels, R. P. J., Leányvári, Z., Csiba, L.
Dátum:1999
ISSN:0940-5429
Megjegyzések:Blood glucose and insulin concentrations have been reported to influence cerebral hemodynamics. We studied the relationship between actual blood glucose and insulin concentrations and resting cerebral blood flow velocity in the middle cerebral artery and cerebrovascular reserve capacity after acetazolamide stimulation. Thirty-six insulin-dependent diabetic patients in a state of good glycemic control were studied. Blood samples were taken for determination of glucose and insulin concentrations. Subsequently we measured resting cerebral blood flow velocities in supine position using transcranial Doppler, administered 1 g acetazolamide intravenously, and repeated the measurements after 5, 10, 15 and 20 minutes. Cerebrovascular reserve was calculated as the maximal percent increase after acetazolamide stimulation. Multiple regression was used for statistical analysis. Blood glucose levels were not correlated with resting blood flow velocity (R = 0.21, p = 0.22) nor cerebrovascular reserve capacity (R = 0.17, p = 0.32). Similarly, no correlation was found between insulin concentrations, resting cerebral blood flow velocity (R = 0.24, p = 0.22) and cerebrovascular reserve (R = 0.26, p = 0.24). Studying patients with long-term (> 10 years) and short-term (</= 10 years) disease duration yielded the same lack of correlation. We conclude that there is no significant correlation between contemporaneously measured glucose and insulin concentrations and either cerebral blood flow velocity or cerebrovascular reserve capacity in the middle cerebral artery in type 1 diabetic patients with good control.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Acta Diabetologica. - 36 : 4 (1999), p. 191-195. -
További szerzők:Limburg, Martien Bereczki Dániel (1960-) (neurológus) Molnár Csilla (1962-) (aneszteziológus) Michels, R. P. J. Leányvári Zsolt (1968-) (neurológus) Csiba László (1952-) (neurológus, pszichiáter)
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4.

001-es BibID:BIBFORM042395
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Impairment of cerebrovascular reactivity in long-term type 1 diabetes / Fulesdi, B., Limburg, M., Bereczki, D., Michels, R. P., Neuwirth, G., Legemate, D., Valikovics, A., Csiba, L.
Dátum:1997
ISSN:0012-1797
Megjegyzések:The early preclinical detection of cerebrovascular complications in individuals with diabetes is one of the goals of care described in the St. Vincent Declaration. In accordance with this goal, the aim of the present work was to investigate whether altered cerebral microvascular function in patients suffering from type 1 diabetes can be detected with a transcranial Doppler probe after the administration of acetazolamide. A total of 72 type 1 diabetic patients and 40 healthy control subjects entered the study. Patients were divided into two groups: those with long-term diabetes (disease duration of >10 years, n = 37) and those with short-term diabetes (disease duration of < or =10 years, n = 35). Mean blood-flow velocity in the middle cerebral artery (MCAV) was measured at rest and at 5, 10, 15, and 20 min after intravenous administration of 1 g acetazolamide with a transcranial Doppler probe and expressed as the percentage change from the pretest measurement. The percentage increase in MCAV (cerebrovascular reactivity) was calculated at each time point and compared between the groups. Cerebrovascular reserve capacity (CRC), expressed as the maximal percentage increase of the MCAV, was compared between the groups. Additionally, a reproducibility study of CRC was performed in 10 patients, using intraclass correlations. Cerebrovascular reactivity in the long-term diabetes group was lower (means +/- SD: 5 min, 23.4 +/- 15.4%; 10 min, 28.8 +/- 17.0%; 15 min, 30.0 +/- 15.6%; 20 min, 24.2 +/- 17.8%) than that of the control subjects (5 min, 43.5 +/- 23.9%; 10 min, 55.3 +/- 24.0%; 15 min, 56.7 +/- 23.8%; 20 min, 54.8 +/- 25.9%) and the short-term diabetic patients (5 min, 43.6 +/- 25.9%; 10 min, 52.2 +/- 27.7%; 15 min, 55.3 +/- 32.2%; 20 min, 45.8 +/- 35.8%). CRC was lower in the long-term diabetes group than in the control group or the short-term diabetes group. Impairment of cerebrovascular reactivity was associated with retino- and nephropathy and increased levels of fibrinogen. In contrast, CRC was independent from actual glucose, insulin, glycosylated hemoglobin, von Willebrand factor antigen, and alpha-2 macroglobulin levels. Transcranial Doppler measurements of the changes in MCAV after stimulation with acetazolamide can detect altered cerebral microvascular function in patients with diabetes. Cerebrovascular reactivity and reserve capacity are reduced in patients with long-term diabetes. Further prospective studies should delineate the clinical significance of our results.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Diabetes. - 46 : 11 (1997), p. 1840-1845. -
További szerzők:Limburg, Martien Bereczki Dániel (1960-) (neurológus) Michels, R. P. J. Neuwirth Gyula Legemate, D. Valikovics Attila Csiba László (1952-) (neurológus, pszichiáter)
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5.

001-es BibID:BIBFORM033458
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Lack of gender difference in acetazolamide-induced cerebral vasomotor reactivity in patients suffering from type-1 diabetes mellitus / Fulesdi, B., Limburg, M., Olah, L., Bereczki, D., Csiba, L., Kollar, J.
Dátum:2001
Megjegyzések:The aim of the present work was to investigate the impact of gender on resting cerebral blood flow velocity and cerebrovascular reserve capacity among diabetic patients. Middle cerebral artery mean blood flow velocity (MCAV) was measured in 72 patients suffering from type 1 diabetes mellitus at rest and 5, 10, 15 and 20 min after intravenous administration of 1 g acetazolamide. Cerebrovascular reserve was calculated as the maximal percent increase in MCAV after acetazolamide. Resting MCAV and cerebrovascular reserve capacity were compared between males and females. Resting cerebral blood flow velocity was higher in diabetic females than in males (men, 55.0+/-17.0 cm/s; women, 64.4+/-12.6 cm/s, p=0.0094). Cerebrovascular reserve capacity was similar in diabetic women and men (men, 44.0%+/-18.6%; women, 52.6%+/-32.9%, p=0.17). Comparing MCAV and cerebrovascular reserve capacity among the diabetic subgroups with disease duration < or = 10 years and >10 years, we did not detect any differences between women and men. Duration of diabetes was an important factor in determining cerebrovascular reserve capacity in both sexes: long-term diabetic women and men showed lower CRC values than diabetics with < or = 10 years disease duration. Cerebrovascular reserve capacity is similar in diabetic women and men. Taking into consideration that cerebrovascular reserve is normally higher among women, our finding indicates a relatively more serious worsening of cerebral vasodilatory responses in women suffering from type 1 diabetes.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Acta Diabetologica. - 38 : 3 (2001), p. 107-112. -
További szerzők:Limburg, Martien Oláh László (1967-) (neurológus) Bereczki Dániel (1960-) (neurológus) Csiba László (1952-) (neurológus, pszichiáter) Kollár József (1950-) (radiológus)
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6.

001-es BibID:BIBFORM029158
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Cerebrovascularis reaktivitás vizsgálata az arteria carotisok tünetmentes és tünetet okozó atheroscleroticus laesioiban szenvedő betegekben / Fülesdi B., Valikovics A., Orosz L., Oláh L., Limburg M., Dink L., Káposzta Z., Csiba L.
Dátum:1998
Tárgyszavak:Orvostudományok Klinikai orvostudományok magyar nyelvű folyóiratközlemény hazai lapban
Megjelenés:Orvosi Hetilap. - 139 : 11 (1998), p. 623-628. -
További szerzők:Valikovics Attila Orosz László (1966-) (sebész) Oláh László (1967-) (neurológus) Limburg, Martien Dink, Legemate Káposzta Zoltán Csiba László (1952-) (neurológus, pszichiáter)
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7.

001-es BibID:BIBFORM023401
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Cerebrovascular reactivity and reserve capacity in type II diabetes mellitus / Fülesdi B., Limburg M., Bereczki D., Káplár M., Molnár C., Kappelmayer J., Neuwirth G., Csiba L.
Dátum:1999
ISSN:1056-8727
Megjegyzések:The aim of the study was to test the hypothesis that cerebrovascular reserve capacity and cerebrovascular reactivity are impaired in patients suffering from non insulin-dependent diabetes mellitus. We also intended to investigate factors which may influence resting cerebral blood flow velocity and cerebrovascular reserve capacity. A total of 28 patients suffering from type II diabetes mellitus and 20 healthy control subjects were studied. Based on diabetes duration patients were divided into two groups: subjects with > 10 years and those with < or = 10 years disease duration. Middle cerebral artery mean blood flow velocities were measured at rest and after intravenous administration of 1g acetazolamide. Cerebrovascular reactivity and reserve capacity were calculated. Blood glucose, insulin, glycosylated hemoglobin, hemostatic factors (fibrinogen, alpha-2 macroglobulin and von Willebrand factor antigen) were determined. Cerebrovascular reactivity and reserve capacity values were compared between the two diabetic subgroups and controls. Correlations between laboratory parameters and cerebrovascular reserve were investigated by linear regression analysis. Resting cerebral blood flow velocity was similar in controls and in the two diabetic subgroups. Cerebrovascular reactivity was elevated for a shorter time in patients with > 10 years disease duration than in controls and short-term diabetic patients. Cerebrovascular reserve capacity was lower in the long-term diabetes group (means +/- SD: 39.6 +/- 20.7%) than in patients with < or = 10 years disease duration (63.3 +/- 17.4%, p < 0.02 after Bonferroni correction). Cerebrovascular reserve capacity was inversely related to the duration of the disease (R = 0.53, p < 0.003). None of the determined laboratory factors had any relation with resting cerebral blood flow and cerebrovascular reserve capacity. The vasodilatory ability of cerebral arterioles is diminished in long-standing type II diabetes mellitus.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
egyetemen (Magyarországon) készült közlemény
Megjelenés:Journal Of Diabetes And Its Complications. - 13 : 4 (1999), p. 191-199. -
További szerzők:Limburg, Martien Bereczki Dániel (1960-) (neurológus) Káplár Miklós (1965-) (belgyógyász, diabetológus) Molnár Csilla (1962-) (aneszteziológus) Kappelmayer János (1960-) (laboratóriumi szakorvos) Neuwirth Gyula Csiba László (1952-) (neurológus, pszichiáter)
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8.

001-es BibID:BIBFORM023979
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Cerebrovascularis reaktivitás nem inzulindependens diabetes mellitusban : előzetes eredmények / Fülesdi, B., Limburg, M., Molnár, Cs., Káplár, M., Bereczki, D., Neuwirth, Gy., Csiba, L.
Dátum:1998
Tárgyszavak:Orvostudományok Klinikai orvostudományok magyar nyelvű folyóiratközlemény hazai lapban
egyetemen (Magyarországon) készült közlemény
Megjelenés:Orvosi Hetilap. - 139 : 30 (1998), p. 1789-1792. -
További szerzők:Limburg, Martien Molnár Csilla (1962-) (aneszteziológus) Káplár Miklós (1965-) (belgyógyász, diabetológus) Bereczki Dániel (1960-) (neurológus) Neuwirth Gyula Csiba László (1952-) (neurológus, pszichiáter)
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9.

001-es BibID:bibEBI16618
Első szerző:Fülesdi Béla (aneszteziológus)
Cím:Evaluation of the cerebrovascular reserve capacity in asymptomatic and symptomatic carotid artery abstructive disease / Fülesdi, B., Káposzta, Z., Molnár, Cs., Oláh, L., Limburg, M., Valikovics, A., Csiba, L.
Dátum:1995
Tárgyszavak:Orvostudományok Klinikai orvostudományok idézhető absztrakt
egyetemen (Magyarországon) készült közlemény
Megjelenés:European Journal of Neurology. - 2 : Suppl.2 (1995), p. 21. -
További szerzők:Káposzta Zoltán Molnár Csilla (1962-) (aneszteziológus) Oláh László (1967-) (neurológus) Limburg, Martien Valikovics Attila Csiba László (1952-) (neurológus, pszichiáter)
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10.

001-es BibID:BIBFORM023404
Első szerző:Hidasi Eszter (neurológus)
Cím:No correlation between impairment of cerebrovascular reserve capacity and electrophysiologically assessed severity of neuropathy in noninsulin-dependent diabetes mellitus / Hidasi E., Káplár M., Diószeghy P., Bereczki D., Csiba L., Limburg M., Fülesdi B.
Dátum:2002
Megjegyzések:Microvascular abnormalities have an important role in the most frequent neurological complications of diabetes mellitus: neuropathy and cerebrovascular disorders. Severity of neuropathy as well as of cerebral microvascular damage can be quantitatively evaluated by instrumental methods like nerve conduction studies and transcranial Doppler. In the present study, we investigated whether a correlation exists between the severity of peripheral neuropathy and the impairment of cerebrovascular reserve capacity (CRC) in 20 patients with Type 2 diabetes mellitus. METHODS: CRC was measured by transcranial Doppler and defined as the maximal percentage increase in blood flow velocity in the middle cerebral artery within 20 min after an intravenous dose of 1000 mg of acetazolamide. Nerve conduction studies of the median, ulnar, peroneal, and sural nerves were performed. Severity of neuropathy was scored based on conduction velocities, amplitudes, and distal latencies. RESULTS: There was no correlation between the neuropathic score and CRC (R= .003, P= .99). Neither CRC nor the neuropathic score correlated significantly with age, duration of diabetes, and serum values of HbA(1c), glucose, insulin, von Willebrand factor, and alpha(2) - macroglobulin. Severity of neuropathy but not CRC correlated with microalbuminuria (R= .47, P= .038 and R= .14, P= .54). Improper treatment reflected by HbA(1c) >10% was associated with significantly more severe albuminuria, higher actual blood glucose level, higher von Willebrand factor activity, and marginally higher neuropathic score (21 vs. 13, P=.096), but was not associated with CRC (44% vs. 42%, P= .81). When duration of diabetes was dichotomized to 15 years and less or over 15 years, CRC was significantly smaller (35% vs. 50%, P= .036) and neuropathy was more severe in the subgroup with longer diabetes duration (19 vs. 11.5 points, P= .07). CONCLUSIONS: Although both CRC and peripheral nerve function are affected more severely in patients with long-lasting Type 2 diabetes mellitus, damage in the cerebrovascular system and in the long peripheral nerves occur independently. As in diabetes mellitus pathological changes in autonomic and large peripheral nerves develop simultaneously, decreased CRC in diabetic patients might be predominantly due to structural changes of resistance arteries or to metabolic than to neurogenic factors.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
Megjelenés:Journal of Diabetes and its Complications. - 16 : 3 (2002), p. 228-234. -
További szerzők:Káplár Miklós (1965-) (belgyógyász, diabetológus) Diószeghy Péter (1948-) (ideg- és elmeszakorvos) Bereczki Dániel (1960-) (neurológus) Csiba László (1952-) (neurológus, pszichiáter) Limburg, Martien Fülesdi Béla (1961-) (aneszteziológus)
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11.

001-es BibID:BIBFORM029162
Első szerző:Orosz László (sebész)
Cím:Assessment of cerebrovascular reserve capacity in asymptomatic and symptomatic hemodynamically significant carotid stenoses and occlusions / László Orosz, Béla Fülesdi, Arjan Hoksbergen, Georgios Settakis, József Kollár, Martien Limburg, György Csécsei
Dátum:2002
ISSN:0090-3019
Megjegyzések:Cerebrovascular reactivity measurements are believed to be a helpful tool for selecting patients who are at higher risk for hemodynamic strokes. The aim of this study was to compare cerebral vasoreactivity among patients suffering from internal carotid artery stenosis of different severity (asymptomatic stenosis, asymptomatic occlusion, symptomatic stenosis, symptomatic occlusion). METHODS: Sixty-two patients with asymptomatic and symptomatic internal carotid artery stenoses and occlusions underwent transcranial Doppler-acetazolamide tests. Absolute velocities of the middle cerebral arteries (MCAV), percent increases of the MCAV at different time points of the test (cerebrovascular reactivity, CVR) and maximal percent increase after administration of acetazolamide (cerebrovascular reserve, CRC) were compared on the affected and non-affected sides. Asymmetry indices (CRC (affected side)/CRC (non-affected side)) were compared between the groups of different severity of obstructive lesion. RESULTS: Resting MCAV was similar on both sides in all groups. A significant side-difference of the MCAV values after acetazolamide was observed only in the symptomatic groups. Difference of cerebrovascular reserve capacity between the affected and non-affected side was statistically significant only in the symptomatic groups (CRC symptomatic stenosis 36.6 +/- 20.9% vs. 71.1 +/- 27.9%, CRC symptomatic occlusion: 31.2 +/- 24.6% vs. 64.5 +/- 29.7%). Asymmetry index of the CRC was near to 1 in the asymptomatic stenosis group only, while in all the other groups this index referred to a significant hemispheric asymmetry of the vasoreactivity. CONCLUSIONS: Although in general cerebrovascular reserve capacity is compromised in cases of hemodynamically significant carotid lesions, there is a large individual variability within the subgroups. Further randomized studies are needed to clarify whether the clinical efficiency of carotid endarterectomy and extra-intracranial bypass may be improved by selecting the patients using hemodynamic criteria.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
egyetemen (Magyarországon) készült közlemény
Megjelenés:Surgical Neurology. - 57 : 5 (2002), p. 333-339. -
További szerzők:Fülesdi Béla (1961-) (aneszteziológus) Hoksbergen, Arjan W. J. Settakis, George Kollár József (1950-) (radiológus) Limburg, Martien Csécsei György (1948-2005) (idegsebész)
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12.

001-es BibID:BIBFORM025492
Első szerző:Páll Dénes (belgyógyász, kardiológus)
Cím:Angiotensin-converting enzyme gene polymorphism, carotid intima-media thickness, and left ventricular mass index in adolescent hypertension / Dénes Páll, Georgios Settakis, Éva Katona, János Zatik, József Kollár, Martien Limburg, Béla Fülesdi
Dátum:2004
Megjegyzések:The aim of this study was to determine whether a positive correlation exists between the DD genotype of angiotensin-converting enzyme (ACE) and the carotid intima-media thickness (IMT) or left ventricular mass index (LVMI) in adolescents with hypertension. METHODS: We studied 120 hypertensive and 58 normotensive (control) adolescents. ACE gene polymorphism was determined by a standardized method. The IMT was measured in the common carotid arteries, and the LVMI was calculated on transthoracic echocardiography. The severity of hypertension was graded according to the results of ambulatory blood pressure monitoring as white coat, borderline, or proven hypertension. The association between ACE gene polymorphism and the obtained parameters was analyzed using analysis of variance and chi-square testing. RESULTS: Both the IMT and LVMI were higher in hypertensive than in control adolescents. In hypertensive subjects, no relationship was found between the different ACE genotypes and the IMT: the mean (+/- standard deviation) IMT in patients with the DD genotype was 0.57 +/- 0.11 mm; in those with the DI genotype, 0.53 +/- 0.01 mm; and in those with the II genotype, 0.55 +/- 0.01 mm. The LVMI was also similar in all 3 ACE genotypes: DD, 106.7 +/- 32.3 g/m(2); DI, 102.2 +/- 38.1 g/m(2); and II, 103.2 +/- 26.3 g/m(2). The severity of hypertension as assessed by ambulatory blood pressure monitoring was independent of the ACE genotype. CONCLUSIONS: Target-organ damage (left ventricular hypertrophy and carotid atherosclerosis) is detectable in adolescents with hypertension and, in those subjects, ACE genotype polymorphism is not correlated with the severity of target-organ damage as measured by IMT and LVMI.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
egyetemen (Magyarországon) készült közlemény
Megjelenés:Journal of clinical ultrasound. - 32 : 3 (2004), p. 129-135. -
További szerzők:Settakis, George Katona Éva (1967-) (belgyógyász, diabetológus) Zatik János (1961-) (szülész-nőgyógyász) Kollár József (1950-) (radiológus) Limburg, Martien Fülesdi Béla (1961-) (aneszteziológus)
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