CCL

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1.

001-es BibID:BIBFORM041391
035-os BibID:PMID:7691934
Első szerző:Ádány Róza (megelőző orvostan és népegészségtan szakorvos)
Cím:Arborizing tenascin network under the rapidly proliferating epithelium of epulis / Róza Ádány, Makoto Toida, Tsuyoshi Takami
Dátum:1993
Tárgyszavak:Orvostudományok Egészségtudományok szerkesztői levél
egyetemen (Magyarországon) készült közlemény
Megjelenés:Journal of Histochemistry & Cytochemistry. - 41 : 11 (1993), p. 1717-1718. -
További szerzők:Toida, Makoto Takami, Tsuyoshi
Internet cím:Szerző által megadott URL
DOI
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2.

001-es BibID:BIBFORM041550
035-os BibID:PMID:7558894
Első szerző:Toida, Makoto
Cím:Characterization of cells containing factor XIII subunit a in benign and malignant buccal lesions / M. Toida, Y. Okumura, K. K. Swe Win, N. Oka, T. Takami, R. Ádány
Dátum:1995
Megjegyzések:In the present study, the distribution pattern and characteristics of cells containing Factor XIII subunit a (FXIII A) have been studied in benign and malignant lesions of human buccal mucosa. Tissues from four irritation fibromas and three squamous cell carcinomas were studied by means of double immunofluorescent staining techniques in which the detection of FXIII A was combined with a reaction with CD14 (recognizing a monocyte/macrophage differentiation marker antigen), Mac 387 (reacting with a special subset of macrophages), anti-HLA-DR, Ki-M7 (labelling phagocytosing macrophages) or Ki-67 (visualizing a nuclear antigen associated with cell proliferation) monoclonal antibodies. FXIII A was detected in cells of the connective tissue stroma in both benign and malignant buccal lesions. The number of these FXIII A-reactive cells (FXIII A+ cells) increased considerably in the tumour tissues, in particular in those surrounding tumour cell clusters. FXIII A+ cells scattered in the fibromatous tissues were spindle-shaped, whereas in the tumour stroma, large stellate cells predominated, and round cells were likewise labelled around blood vessels. FXIII A+ cells were labelled with CD14 and Ki-M7 in both fibromatous and tumoural buccal mucosa; however, they failed to show any reaction with Ki-67. FXIII A+ cells accumulated in the tumour stroma reacted for HLA-DR as well. These results indicate that in both the benign and malignant buccal lesions FXIII A is contained in a subpopulation of tissue macrophages, which represents a monocyte-derived (CD14+) and phagocytosing (Ki-M7+) cell population. The accumulation of the FXIII A+ cells in the tumour stroma is believed to be a result of direct migration from the circulating blood.
Tárgyszavak:Orvostudományok Egészségtudományok idegen nyelvű folyóiratközlemény külföldi lapban
külföldön készült közlemény
Megjelenés:Histochemical Journal. - 27 : 6 (1995), p. 449-456. -
További szerzők:Okumura, Y. Swe Win, K. K. Oka, N. Takami, Tsuyoshi Ádány Róza (1952-) (megelőző orvostan és népegészségtan szakorvos)
Internet cím:Intézményi repozitóriumban (DEA) tárolt változat
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3.

001-es BibID:BIBFORM041553
035-os BibID:PMID:7558893
Első szerző:Toida, Makoto
Cím:Accumulation of cells containing factor XIII subunit a around the foci of intense fibrosis in human epulides / M. Toida, N. Oka, T. Takami, R. Ádány
Dátum:1995
Megjegyzések:On the basis of clinical and biochemical findings, Factor XIII subunit a (FXIII A) has been conjectured to play an important role in fibrotic processes. Epulis samples at different stages of fibrotic tissue formation were used as a model system for studying the localization and tissue distribution of FXIII A during the course of connective tissue generation. Marker characteristics of cells containing FXIII A (FXIII A+ cells) were determined as well. In double immunofluorescent labelling systems, FXIII A was localized in monocyte-derived (CD-14+), activated (HLA-DR+), and phagocytosing (Ki-M7+) tissue macrophages, which are widely distributed homogeneously in granulation tissues, but start to accumulate around foci of fibrosis as soon as the foci appear. During the relatively long process of fibrosis, FXIII A+ macrophages continuously decrease in number, and their morphological appearance changes from stellate to spindle-shaped. The nuclei of these cells were not labelled by Ki-67 monoclonal antibody; this indicating that they represent a non-proliferating cell population in the connective tissue stroma. The present findings may help to link theories concerning the role of FXIII A and those of macrophages in the connective tissue formation so far found separately in the literature.
Tárgyszavak:Orvostudományok Klinikai orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban
külföldön készült közlemény
Megjelenés:Histochemical Journal. - 27 : 6 (1995), p. 440-448. -
További szerzők:Oka, N. Takami, Tsuyoshi Ádány Róza (1952-) (megelőző orvostan és népegészségtan szakorvos)
Internet cím:Intézményi repozitóriumban (DEA) tárolt változat
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4.

001-es BibID:BIBFORM033268
035-os BibID:WOS:000169236100006
Első szerző:Toida, Makoto
Cím:Analysis of genetic alterations in salivary gland tumors by comparative genomic hybridization / Makoto Toida, Margit Balázs, Tomohiro Mori, Jun-Ichi Ishimaru, Hideki Ichihara, Hideki Fujitsuka, Iwao Hyodo, Kyoko Yokoyama, Norichika Tatematsu, Róza Ádány
Dátum:2001
Megjegyzések:In order to define and map chromosomal copy number alterations in salivary gland tumors (SGTs), a comparative genomic hybridization (CGH) technique was applied to two pleomorphic adenomas (PAs), one adenoid cystic carcinoma (ACC), and one basal cell adenocarcinoma (BCAC). The PAs exhibited regional copy number losses at 5q12.4-q14.1. 9q12-q21.13. and 16q11.2, as well as a gain at 20p12.1; among these, the losses at the 9q12-q21.11 and 16q11.2 regions were common to both PAs. The ACC showed overrepresentations of the entire regions of chromosomes 16 and 20, a regional gain at 22q12.3-q13.1, and no losses. In the BCAC. regional gains at 9p21.1-pter, 18q21.1-q22.3, and 22q11.23-q13.31 as well as losses at 2q24.2 and 4q25-q27 were seen; the gain at 22q12.3-q13.1 was common in both the ACC and the BCAC. These CGH data indicate that different genetic alterations are present in the different types of SGTs, and that the alterations involve several chromosomes. The discovery of common alterations in the same and/or different types of tumors might be important in the understanding of the development and progression of the SGTs. (C) 2001 Elsevier Science Inc. All rights reserved.
Tárgyszavak:Orvostudományok Egészségtudományok idegen nyelvű folyóiratközlemény külföldi lapban
külföldön készült közlemény
Megjelenés:Cancer Genetics and Cytogenetics. - 127 : 1 (2001), p. 34-37. -
További szerzők:Mori, Tomohiro Ishimaru, Jun-Ichi Ichihara, Hideki Fujitsuka, Hideki Hyodo, Iwao Yokoyama, Kyoko Tatematsu, Norichika Balázs Margit (1952-) (sejtbiológus, molekuláris genetikus) Ádány Róza (1952-) (megelőző orvostan és népegészségtan szakorvos)
Internet cím:DOI
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