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001-es BibID:	BIBFORM106092
035-os BibID:	(cikkazonosító)16 (WOS)000909939400001 (Scopus)85145967685
Első szerző:	Türk-Mázló Anett (molekuláris biológus)
Cím:	Resolution Potential of Necrotic Cell Death Pathways / Anett Mázló, Yidan Tang, Viktória Jenei, Jessica Brauman, Heba Yousef, Attila Bácsi, Gábor Koncz
Dátum:	2022
ISSN:	1422-0067
Megjegyzések:	Abstract: During tissue damage caused by infection or sterile inflammation, not only damage-associated molecular patterns (DAMPs), but also resolution-associated molecular patterns (RAMPs) can be activated. These dying cell-associated factors stimulate immune cells localized in the tissue environment and induce the production of inflammatory mediators or specialized proresolving mediators (SPMs). Within the current prospect of science, apoptotic cell death is considered the main initiator of resolution. However, more RAMPs are likely to be released during necrotic cell death than during apoptosis, similar to what has been observed for DAMPs. The inflammatory potential of many regulated forms of necrotic cell death modalities, such as pyroptosis, necroptosis, ferroptosis, netosis, and parthanatos, have been widely studied in necroinflammation, but their possible role in resolution is less considered. In this review, we aim to summarize the relationship between necrotic cell death and resolution, as well as present the current available data regarding the involvement of certain forms of regulated necrotic cell death in necroresolution.
Tárgyszavak:	Orvostudományok Elméleti orvostudományok idegen nyelvű folyóiratközlemény külföldi lapban folyóiratcikk
Megjelenés:	International Journal Of Molecular Sciences. - 24 : 1 (2022), p. 1-24. -
További szerzők:	Tang, Yidan Jenei Viktória (1997-) (biológus) Brauman, Jessica Mustafa, Heba Yousef Hussein (2000-) (orvos) Bácsi Attila (1967-) (immunológus) Koncz Gábor (1970-) (biológus, immunológus)
Pályázati támogatás:	ÚNKP-22-3-I-DE-97 ÚNKP NTP-NFTÖ-21-B-0187, NTP-NFTÖ-22-B-0129 Egyéb GINOP-2.3.2-15-2016-00050 GINOP PD 142930 NKFIH
Internet cím:	DOI Intézményi repozitóriumban (DEA) tárolt változat
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